The presentation must educate advanced practice nurses on assessment and . It has a simple style, with a white background and light blue waves and lines, which convey elegance and serenity. Considerations Problems other than COLD should be suspected when hypoxemia is difficult to correct with modest levels of supplemental oxygen. welcome to the literature of the year. In addition, we have included a multitude of resources that you can edit to convey your information, such as graphics, map, infographics, etc. Nursing Times [online]; 116: 4, 27-30. . NUR. islamic university of science and technology, COPD (Chronic obstructive Pulmonary Disease) PowerPoint Presentation -aslam, Nursing management of the client with increased intracranial pressure, Upper respiratory disorders and nursing mangement, Chronic obstructive pulmonary disease (copd) power point, COPD(chronic obstructive pulmonary disease) ppt slideshare, GOLD16 MANAGEMENT AND TREATMENT OF COPD, Emphysema-medical information |management |diagnosis | tests. By accepting, you agree to the updated privacy policy. working around certain kinds of chemicals, COPD - . review oct. 16, 2014 cathy vakil. The patient and caregiver track contains several helpful tools, including an interactive My COPD Action Plan and activity tracking calendar; inhaler, nebulizer and exercise videos; and a wallet card to track important information such as medications and immunizations. At the end of the end of the 16th International Family Nursing Conference, attendees should be able to discuss global advancements in family nursing education for students and professionals. experiencing shortness of breath at rest or with minimal activity, such as walking from one room to another. Free access to premium services like Tuneln, Mubi and more. Epidemiology >70% of COLD-related health care expenditures go to emergency department visits and hospital care (>$10 billion annually in the U.S.). 1) suspect copd - prolonged or recurrent cough, dyspnea, or, COPD - . possible areas to cover. Art of Assertive Commnunication, how to be visible in social media by Shreed UiPath Automation Cloud Robots - Best Practises session 2.pptx, How To Restore Tree From Ancestry To Family Tree Maker, INVERTING BUCK-BOOST DCDC CONVERTER DESIGN CHALLENGES. Do not sell or share my personal information, 1. CPR - Cardiac Pulmonary Resuscitation by completion of introductory period. The inclusion criteria for . Mr. Yahye Sheikh Abdulle Msc Nursing 1st year Kle college of Nursing Chronic obstructive pulmonary diseases 2. Separately COPD is more costly per case than . Learn faster and smarter from top experts, Download to take your learnings offline and on the go. Clipping is a handy way to collect important slides you want to go back to later. Chronic obstructive pulmonary disease, also known as COPD, encompasses a group of diseases that cause problems with breathing. 28-7, Emphysema Pathophysiology Hyperinflation of alveoli Destruction of alveolar walls Destruction of alveolar capillary walls Narrowed airways Loss of lung elasticity, Emphysema Pathophysiology Two types: Centrilobular (central part of lobule) Most common Panlobular (destruction of whole lobule) Usually associated with AAT deficiency, Emphysema Pathophysiology Structural changes are: Hyperinflation of alveoli Destruction of alveolar capillary walls Narrowed, tortuous small airways Loss of lung elasticity, Emphysema Pathophysiology Small bronchioles become obstructed as a result of Mucus Smooth muscle spasm Inflammatory process Collapse of bronchiolar walls Recurrent infections production/stimulation of neutrophils and macrophages release proteolytic enzymes alveolar destruction inflammation, exudate, and edema, Emphysema Pathophysiology Elastin and collagen are destroyed Air goes into the lungs but is unable to come out on its own and remains in the lung Causes bronchioles to collapse, Emphysema Pathophysiology Trapped air hyperinflation and overdistention As more alveoli coalesce, blebs and bullae may develop Destruction of alveolar walls and capillaries reduced surface area for O2 diffusion Compensation is done by increasing respiratory rate to increase alveolar ventilation Hypoxemia usually develops late in disease, Emphysema Clinical Manifestations Dyspnea Progresses in severity Patient will first complain of dyspnea on exertion and progress to interfering with ADLs and rest, Emphysema Clinical Manifestations Minimal coughing with no to small amounts of sputum Overdistention of alveoli causes diaphragm to flatten and AP diameter to increase, Emphysema Clinical Manifestations Patient becomes chest breather, relying on accessory muscles Ribs become fixed in inspiratory position, Emphysema Clinical Manifestations Patient is underweight (despite adequate calorie intake), Chronic Bronchitis Pathophysiology Pathologic lung changes are: Hyperplasia of mucus-secreting glands in trachea and bronchi Increase in goblet cells Disappearance of cilia Chronic inflammatory changes and narrrowing of small airways Altered fxn of alveolar macrophages infections, Chronic Bronchitis Pathophysiology Chronic inflammation Primary pathologic mechanism causing changes Narrow airway lumen and reduced airflow d/t hyperplasia of mucus glands Inflammatory swelling Excess, thick mucus, Chronic Bronchitis Pathophysiology Greater resistance to airflow increases work of breathing Hypoxemia and hypercapnia develop more frequently in chronic bronchitis than emphysema, Chronic Bronchitis Pathophysiology Bronchioles are clogged with mucus and pose a physical barrier to ventilation Hypoxemia and hypercapnia d/t lack of ventilation and O2 diffusion Tendency to hypoventilate and retain CO2 Frequently patients require O2 both at rest and during exercise, Chronic Bronchitis Pathophysiology Cough is often ineffective to remove secretions because the person cannot breathe deeply enough to cause air flow distal to the secretions Bronchospasm frequently develops More common with history of smoking or asthma, Chronic Bronchitis Clinical Manifestations Earliest symptoms: Frequent, productive cough during winter Frequent respiratory infections, Chronic Bronchitis Clinical Manifestations Bronchospasm at end of paroxysms of coughing Cough Dyspnea on exertion History of smoking Normal weight or heavyset Ruddy (bluish-red) appearance d/t polycythemia (increased Hgb d/t chronic hypoxemia)) cyanosis, Chronic Bronchitis Clinical Manifestations Hypoxemia and hypercapnia Results from hypoventilation and airway resistance + problems with alveolar gas exchange, COPD Complications Pulmonary hypertension (pulmonary vessel constriction d/t alveolar hypoxia & acidosis) Cor pulmonale (Rt heart hypertrophy + RV failure) Pneumonia Acute Respiratory Failure, COPD Diagnostic Studies Chest x-rays early in the disease may not show abnormalities History and physical exam Pulmonary function studies reduced FEV1/FVC and residual volume and total lung capacity, COPD Diagnostic Studies ABGs PaO2 PaCO2 (especially in chronic bronchitis) pH (especially in chronic bronchitis) Bicarbonate level found in late stages COPD, COPD Collaborative Care Smoking cessation Most significant factor in slowing the progression of the disease, COPD Collaborative Care: Drug Therapy Bronchodilators as maintenance therapy -adrenergic agonists (e.g. Takeaway. Testing positive for COVID-19 can be scary and overwhelming, and people with COPD are at higher risk for severe symptoms. (due to weakened/impaired physical activity, tiredness, etc) 2.) Cancellation Policy: Mr. ANILKUMAR B R , Lecturer The inclusion criterion for nurses was employment in home nursing care for at least 6 months. Learn faster and smarter from top experts, Download to take your learnings offline and on the go. You can read the details below. The COPD Foundation's My COPD Action Plan should be filled out the first time as part of a discussion with your health care provider, and then used daily or as often as you can. The air sacs fill up with air, like a small balloon. rada jones md 09/12/06. Pneumothorax occurs when the parietal or visceral pleura is breached and the pleural . Chronic obstructive pulmonary disease by aminu arzet, Copd(chronic obstructive pulmonary disease), Chronic Obstructive Pulmonary Disease (COPD), Chronic obstructive pulmonary disease ppt, chronic obstructive pulmonary disease in equine. 21 slides. Normally, the airways and air sacs in your lungs are elastic or stretchy. COPD - Physical Findings Tachypnea Accessory respiratory muscle use Pursed lip exhalation Weight loss due to poor dietary intake and excessive caloric expenditure for work of breathing, Dominant Clinical Forms of COPD Pulmonary emphysema Chronic bronchitis Most patients exhibit a mixture of symptoms and signs, COPD - Advanced Dx secondary polycythemia cyanosis tremor somnolence and confusion due to hypercarbia Secondary pulmonary HTN w or w/o cor pulmonale, COPD Treatment Strategy Elimination of extrinsic irritants bronchodilator & glucocorticoid therapy Antibiotics Mobilization of secretions respiratory vaccines Oxygen therapy - if oxygen saturation <90% at rest on room air, A-a gradient A-a gradient = predicted pO2 observed PO2 PAO2 = (FIO2 X 713) (PaCO2/0.8) at sealevel PAO2 = 150-(PaCO2/0.8) at sealevel on room air Normal range 10-15mm > 30 years of age Normal range 8mm < 30 years of age Increased A-aDO2=diffusion defect Right to left shunt V/Q mismatch, Examples A doubel overdose brings two 30 yr old patients to the ED. Asthma patients can also develop chronic (not fully reversible) airflow obstruction. Provided by: Wad5. And/or Weve updated our privacy policy so that we are compliant with changing global privacy regulations and to provide you with insight into the limited ways in which we use your data. Comment The A-a gradient calculation for patient 2 is as follows: PAO2 = 150 (1.25 x PCO2) PAO2 = 150 (1.25 x 50) PAO2 = 150 63 PAO2 = 87 Therefore, A-a = 87 50 =37 (an abnormally increased gradient). mr. d has. having lower oxygen levels than normal . Nursing management of chronic obstructive pulmonary disease. Chronic Bronchitis Chronic lower airway inflammation Increased bronchial mucus production Productive cough Urban male smokers > 30 years old, Chronic Bronchitis Mucus, swelling interfere with ventilation Increased CO2, decreased 02 Cyanosis occurs early in disease Lung disease overworks right ventricle Right heart failure occurs RHF produces peripheral edema Blue Bloater, Emphysema Loss of elasticity in small airways Destruction of alveolar walls Urban male smokers > 40-50 years old, Emphysema Lungs lose elastic recoil Retain CO2, maintain near normal O2 Cyanosis occurs late in disease Barrel chest (increased AP diameter) Thin, wasted Prolonged exhalation through pursed lips Pink Puffer, COPD Management Oxygen Monitor carefully Some COPD patients may experience respiratory depression on high concentration oxygen Assist ventilations as needed, Diagnostic Approach Initial assessment History and physical examination (Signs & Symptoms) Pulmonary function testing to assess airflow obstruction Radiographic studies, Assessment of exacerbation History Fever Change in quantity and character of sputum ill contacts Associated symptoms Frequency and severity of prior exacerbations, Assessment of exacerbation Physical examination Tachycardia Tachypnea Chest examination Focal findings Air movement Symmetry Presence or absence of wheezing Paradoxical movement of abdominal wall Use of accessory muscles Perioral or peripheral cyanosis Ability to speak in complete sentences Mental status, Radiographic studies Chest radiography focal findings (pneumonia, atelectasis) Arterial blood gases Hypoxemia Hypercapnia Hospitalization recommended for: Respiratory acidosis and hypercarbia Significant hypoxemia Severe underlying disease Living situation not conducive to careful observation and delivery of prescribed treatment, ABG and oximetry Although not sensitive, they may demonstrate resting or exertional hypoxemia. Dr. Kaara Ray B. Calma is a full-time Lecturer in the School of Nursing and Midwifery at Deakin University Australia, and is a Registered Nurse with a PhD completed at the University of Wollongong Australia. In the United States alone it affects about 16 million people. The tri-fold card. Occupational exposures to dust and fumes (e.g., cadmium) Likely risk factors The magnitude of these effects appears substantially less important than the effect of cigarette smoking. RN, BSN, PHN. Tap here to review the details. It can be very helpful to have an action plan to follow when managing your COPD. Weve updated our privacy policy so that we are compliant with changing global privacy regulations and to provide you with insight into the limited ways in which we use your data. Chronic obstructive pulmonary diseases & Nursing care. Goal: Develop a PowerPoint presentation on a cardiovascular/pulmonary disorder/disease discussed in the McCance text. . within 30 Days Required ; The disease affects millions of Americans and is a leading cause of disability and death in the U.S. CODP ( Chronic Obstructive Pulmonary Disease ), Chronic Obstructive Pulmonary Disease (COPD), Chronic obstructive pulmonary disease (copd), Approach to Chronic Obstructive Pulmonary Disease, COPD (CHRONIC PULMONARY OBSTRUCTIVE DISEASE) by SUKHERA, Chronic Obstructive Pulmonary Disease - COPD, Chrnonic Obstructive Pulomonary Disease - Pathophysiology, Chronic obstructive airway disease (coad), Chronic obstructive pulmonary disorder (copd), COPD (Chronic obstructive Pulmonary Disease) PowerPoint Presentation -aslam, COPD (Chronic Obstructive Pulmonary Disease), Chronic obstructive pulmonary disorders COPD, Nursing management of the client with increased intracranial pressure, Upper respiratory disorders and nursing mangement, Copd(chronic obstructive pulmonary disease), J. Parker Emphysema Presentation Powerpoint, COPD - Chronic obstructive pulmonary disease - Aby. Comprises primarily of two related disease- CPR - Cardiac Pulmonary Resuscitation by completion of introductory period. Get powerful tools for managing your contents. By accepting, you agree to the updated privacy policy. . If you have COPD, using less energy with daily tasks can help you have more energy to do more activities during the day. . MORE AMOUNT OF SPUTUM. COPD. Msc Nursing 1st year However, people whose disease progresses to more severe stages . Support groups. COPD 1 / 32. Atrovent), COPD Collaborative Care: Oxygen Therapy O2 therapy Raises PO2 in inspired air Treats hypoxemia Titrate to lowest effective dose, COPD Collaborative Care: Oxygen Therapy Chronic O2 therapy at home Improved prognosis Improved neuropsychologic function Increased exercise tolerance Decreased hematocrit Reduced pulmonary hypertension, COPD Collaborative Care: Respiratory Therapy Breathing retraining Pursed-lip breathing Prolongs exhalation and prevents bronchiolar collapse and air trapping Diaphragmatic breathing Focuses on using diaphragm instead of accessory muscles to achieve maximum inhalation and slow respiratory rate See text re how to teach, COPD Collaborative Care: Respiratory Therapy Huff coughing (Table 28-21) Chest physiotherapy to bring secretions into larger, more central airways Postural drainage Percussion Vibration, Positions for Postural Drainage Fig. Enjoy access to millions of ebooks, audiobooks, magazines, and more from Scribd. CT scans can also be used to screen for lung cancer. The SlideShare family just got bigger. chronic obstructive, COPD - . o Presenting at 50 years of age o Strong family history o Predominant basilar disease o Minimal smoking history o Definitive diagnosis of 1AT deficiency requires PI type determination. 3.) COPD (chronic obstructive pulmonary disease) is a group of lung diseases that make it hard to breathe and get worse over time. COPD PowerPoint Presentation. Chronic obstructive pulmonary disease ppt, Chronic obstructive pulmonary disease by aminu arzet, CODP ( Chronic Obstructive Pulmonary Disease ), COPD - Chronic obstructive pulmonary disease - Aby, Pharmacotherapy of Chronic Obstructive Pulmonary Disease, Chronic Obstructive Pulmonary Disease (COPD). Every COPD patient has an anticipatory care plan which is created by their GP . We've encountered a problem, please try again. 20-23 June 2023 Dublin, Ireland. A CT scan of your lungs can help detect emphysema and help determine if you might benefit from surgery for COPD. Smoking is the most common cause. COPD Nursing Diagnosis and Care Plan. COPD is often evaluated in patients with relevant symptoms and risk factors. COPD Chronic obstructive pulmonary disease (COPD) is a lung ailment that is characterized by a persistent blockage of airflow from the lungs. California University Balance Between Formal Teams Communities of Practice Re California State University Light a Candle or Curse the Darkness.docx, California State University Utilitarianism Kantian Ethics Videos Questions.docx. Study Resources. The NEW! PowerPoint Presentation Last modified by: airflow limitation that is progressive and not fully reversible abnormal, COPD- pulmonary hyperinflation- the diaphragms are at the, Occupational exposures to dust and fumes (e.g., cadmium), Distributions of forced expiratory volume in 1 s. Your hunt can take place anywhere. Goal: Develop a PowerPoint presentation on CONGESTIVE HEART FAILURE (CHF) (Please explain implications on cardiovascular and respiratory system) The presentation must provide information about the incidence, prevalence, and pathophysiology of Congestive Heart Failure to the cellular level. Chronic Obstructive Pulmonary Disease (COPD), COPD Description Characterized by presence of airflow obstruction Caused by emphysema or chronic bronchitis Generally progressive May be accompanied by airway hyperreactivity May be partially reversible, Emphysema Description Abnormal permanent enlargement of the air space distal to the terminal bronchioles Accompanied by destruction of bronchioles, Chronic Bronchitis Description Presence of chronic productive cough for 3 or more months in each of 2 successive years in a patient whom other causes of chronic cough have been excluded, COPD Causes Cigarette smoking Primary cause of COPD*** Clinically significant airway obstruction develops in 15% of smokers 80% to 90% of COPD deaths are related to tobacco smoking > 1 in 5 deaths is result of cigarette smoking, COPD Causes Cigarette smoking Nicotine stimulates sympathetic nervous system resulting in: HR Peripheral vasoconstriction BP and cardiac workload, COPD Causes Cigarette smoking Compounds problems in a person with CAD Ciliary activity Possible loss of ciliated cells Abnormal dilation of the distal air space Alveolar wall destruction Carbon monoxide O2 carrying capacity Impairs psychomotor performance and judgment Cellular hyperplasia Production of mucus Reduction in airway diameter Increased difficulty in clearing secretions, COPD Causes Secondhand smoke exposure associated with: Pulmonary function Risk of lung cancer Mortality rates from ischemic heart disease, COPD Causes Infection Major contributing factor to the aggravation and progression of COPD Heredity -Antitrypsin (AAT) deficiency (produced by liver and found in lungs); accounts for < 1% of COPD cases Emphysema results from lysis of lung tissues by proteolytic enzymes from neutrophils and macrophages, Pathophysiology of Chronic Bronchitis and Emphysema Fig. British Journal of Nursing 17 (21). The COPD Foundation is a nonprofit, tax-exempt charitable organization under Section 501(c)(3) of the Internal Revenue Code. Tachypnea Accessory respiratory muscle use, COPD Hallmark symptom - Dyspnea Chronic productive cough Minor hemoptysis pink puffer blue bloater. Click here to review the details. 270 views. By whitelisting SlideShare on your ad-blocker, you are supporting our community of content creators. *ctda0&?iOcdr0&O@(L%EUxmsy]r@y9`P Slide 1-. Each of the nine Guides for Better Living focuses on one important aspect of life with COPD. Expert Help. Chronic Obstructive Pulmonary Disease or Chronic Obstructive lung disease. The signs and symptoms of COPD occur gradually, and may worsen as the disease progresses. Use the contact form to get in touch EtiologyCOLD Causal relationship between cigarette smoking and development of COLD has been proven: however, the response varies considerably among individuals. COPD- pulmonary hyperinflation- the diaphragms are at the level of the eleventh posterior ribs and appear flat. This guide covers the goals of oxygen therapy, oxygen equipment, safety, travel, and more. Tap here to review the details. juliana tambellini university of pittsburgh. This flyer will give you helpful tips and tools for staying healthy, avoiding illness, and recognizing early warning signs of an infection or flare-up. For health care professionals, hard copies of the COPD Pocket Consultant Guide, including the treatment decision tree, seven COPD severity domains, the CAT, the mMRC Breathlessness Scale, and listings of brand or generic medications are available for download below. A chest X-ray can show emphysema, one of the main causes of COPD. COPD - . To download the free app, please visit the App Store or Google Play. Chronic Bronchitis copd. The NCCN 2023 Nursing Program: Advancing Oncology Nursing will provide oncology nurses with comprehensive and clinically relevant information to optimize patient education and care. When you breathe in, the airways bring air to the air sacs. The app. progressive obstruction over time. Kle college of Nursing joshua jewell. Looks like youve clipped this slide to already. 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